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Publisher Correction: Epigenome-wide analysis of T-cell large granular lymphocytic leukemia identifies BCL11B as a potential biomarker

The Original Article was published on 14 November 2022

Correction to: Clinical Epigenetics (2022) 14:148

Following publication of the original article [1], the author noticed an error in the figure. In the published version, the image of Fig. 5 has been repeated twice. The author has informed to remove the duplicated image during correction process. The typesetter has inadvertently missed to correct the figure. However, the text citation and caption of the figure seem to be correct. The corrected Fig. 5 has been published with this erratum.

Fig. 5
figure 5

Interaction of the gene BCL11B with other genes in CD8+ cells. BCL11B long-range interactions from Promoter Capture Hi-C for total CD8-positive T cells (red: BCL11B–C14orf64 (LINC01550) interactions). Genomic regions are depicted in blue circles (promoter region) or blue square (enhancer region)

The original has been corrected.


  1. Bigouret V, Hoffmann T, Arlettaz L, Villard J, Colonna M, Ticheli A, et al. Monoclonal T-cell expansions in asymptomatic individuals and in patients with large granular leukemia consist of cytotoxic effector T cells expressing the activating CD94:NKG2C/E and NKD2D killer cell receptors. Blood. 2003;101:3198–204.

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Correspondence to Patricia Johansson or Teresa Laguna.

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Johansson, P., Laguna, T., Ossowski, J. et al. Publisher Correction: Epigenome-wide analysis of T-cell large granular lymphocytic leukemia identifies BCL11B as a potential biomarker. Clin Epigenet 15, 3 (2023).

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