Fig. 4From: Abnormal expression of PRKAG2-AS results in dysfunction of cardiomyocytes through regulating PRKAG2 transcription by interacting with PPARGKnockdown of PRKAG2b and PRKAG2d resulted in apoptosis of cardiomyocytes. A-B The expression of PRKAG2b and PRKAG2d was significantly reduced by siRNAs. C-D Knocking down PRKAG2b and PRKAG2d resulted in a slight increase in apoptosis of cardiomyocytesBack to article page