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Fig. 5 | Clinical Epigenetics

Fig. 5

From: Neonatal exposure to hyperoxia leads to persistent disturbances in pulmonary histone signatures associated with NOS3 and STAT3 in a mouse model

Fig. 5

H3K9 acetylation at the NOS3 gene locus after TSA treatment. HAEC were exposed to either trichostatin A (TSA) twice for 6 h with and interval of 60 h (light gray) or once for 72 h. Histone modification was analyzed using CHiP immediately after the TSA exposure and compared to unexposed HAEC (black). All experiments were performed in triplicates. Error bars indicated 95% CI. Asterisks indicate significant differences (p < 0.05)

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