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Table 4 ANKRD26 gene expression in relation to metabolic and inflammatory parameters. Relationship between metabolic or inflammatory parameters and ANKRD26 gene expression was assessed in lean and obese individuals by covariate-adjusted Spearman’s rank-order correlation adjusted for age. Number of individuals (n), correlation coefficient r, and age-adjusted p value are shown in the table. TG, triglyceride; TC, total cholesterol; HDL-C, high-density lipoprotein cholesterol; LDL-C, low-density lipoprotein cholesterol; CRP, C-reactive protein; IL, interleukin; IFNγ, interferon γ; TNFα, tumor necrosis factor α; IP-10, interferon gamma-induced protein 10; MCP1, monocyte chemotactic protein 1; MIP1, macrophage inflammatory protein 1; RANTES, regulated on activation, normal T cell expressed, and secreted

From: Epigenetic silencing of the ANKRD26 gene correlates to the pro-inflammatory profile and increased cardio-metabolic risk factors in human obesity

ParametersGene expression
nrp value
Glucose34− 0.233n.s.
TG34− 0.630< 0.001
TC34− 0.113n.s.
HDL-C340.647< 0.001
LDL-C34− 0.315n.s.
CRP34− 0.4940.001
IL-1β30− 0.4980.010
IL-630− 0.4010.027
IL-730− 0.185n.s.
IL-930− 0.123n.s.
IL-1230− 0.5060.002
IL-1730− 0.085n.s.
IFN-γ30− 0.112n.s.
TNF-α30− 0.037n.s.
IL-830− 0.650< 0.001
Eotaxin30− 0.116n.s.
IP-1030− 0.5200.002
MCP-130− 0.206n.s.
MIP-1α30− 0.5130.006
MIP-1β30− 0.601< 0.001
RANTES30− 0.4760.002